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5%) and 22 (34.9%) cases, respectively. On the other hand, based on the Geneva scoring system, moderate and high probabilities were detected in 52 (82.5%) and 10 (15.9%) cases, respectively. Massive PE was present in 21 (33.3%) of PE patients. There was a statistically significant difference in terms of hospital length of stay, mean value of MPV, CTPAOI, CTPAOIR and sPAP in addition to systolic arterial pressure between massive and submassive PE patients (P?Protein Tyrosine Kinase inhibitor (Table?5). The mean D-Dimer levels was positively correlated with CTPAOIR (r?=?0.294, P?=?0.02). Itraconazole There was not a relationship between the mean RDW levels and CTPAOIR (r?=??0.022, P?=?0.865) (Table?5). Platelet count was not correlated with CTPAOIR (r?=??0.28, P?=?0.158) (Table?5). The mean value of MPV levels was positively correlated with the mean value of CTPAOIR (r?=?0.428, P?selleck inhibitor as soon as possible before patient is deceased [15-17]. In the present study, we examined the predictor role of platelet indices and computed tomography scores in the determination of acute PE severity (massive/submassive), and the main finding was the presence of relationships between CTPAOIR and platelet indices with PE severity. There are many studies to determine the relationship between the severity of PE and the radiological scoring systems evaluated with the digital subtraction pulmonary angiograms (Miller index) and computerised tomography (4, 5, 18�C22). Qanadli et?al. [5] proposed a computed tomography index that was correlated with clinical outcomes, like systolic blood pressure, echocardiographic findings and Miller index.