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We enrolled 126 consecutive patients (66 M, 60 F) aged Selleck Obeticholic Acid isomerism, abnormalities in atrioventricular connections), surgery or invasive intervention preceding the onset of arrhythmia, known genetic or familial background of arrhythmias, as well as, systemic disease (muscular dystrophy, juvenile rheumatoid arthritis, glycogen storage diseases, thyroid abnormalities). Children were excluded from the study if they had acute infection, diabetes, elevated blood pressure, electrolyte imbalance or were taking any medications on a regular basis. The control group comprised of 37 (21 M, 16 F) healthy children matched for age, sex and body mass index (BMI), recruited at the same time in the same centre. The Ethical Committee of the Medical University of Silesia in Katowice approved the study (Ref No. KNW-06501-25/I/08), and written, informed consent was obtained from parent and each patient Doxorubicin purchase aged 14 years or more. The diagnosis of arrhythmia was established using the Reynolds Holter 24 hours monitoring system. Based on the type of arrhythmia the study group was divided into two subgroups. The first subgroup consisted of patients with ventricular arrhythmias (single ventricular extrasystoles, incidents of couplets, triplets, ventricular tachycardia, ventricular flutter or fibrillation) while the second group included supraventricular arrhythmias (supraventricular extrasystoles, supraventricular tachycardia and atrial flutter or AF). The FARP1 study group was also divided based on the number of cardiac arrhythmias found in 24 hours Holter monitoring: less than 1000/24 hours, 1-10,000/24 hours, and more than 10,000/24 hours. We also created a separate subgroup for atrial or ventricular tachycardia. We performed transthoracic echocardiography using a Philips 7500 machine with 3-7MHz transducers to exclude congenital heart defects. High-sensitivity CRP was measured by an immunoturbidimetric assay (Dade Behring Marburg GmbH, Marburg, Germany). A commercially available immunoenzymatic assay (R&D Systems, Minneapolis, USA) was used to determine serum IL-6 levels. The minimal detectable IL-6 concentration was 0.03 pg/ml. All the measurements were performed by technicians blinded to the origin of the samples. Intra-assay and inter-assay coefficients of variation for all laboratory variables were