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?4). Patients' demographics are described in Table?1 demonstrating that the study cohort (n?=?69) had similar baseline characteristics to the complete EBV-treated cohort (n?=?96) (Table?1). As previously described, 51 out of the 80 patients who underwent Chartis measurement were prospectively classified as CV?, and 29 were classified as CV+. Regarding the study population of 69 patients, 44 were classified as CV?, and 25 were classified as CV+ (Table?2). The TLVR results showed that 31 of the 44 CV? patients had TLVR that exceeded the 350?mL study threshold. For the CV+ patients, 20 of 25 patients had TLVR Cell Cycle inhibitor Sitaxentan Twenty-seven out of the 34 patients with a complete fissure had TLVR ��350?mL; 26 out of 35 patients with an incomplete fissure had TLVR patients who would achieve TLVR ��350?mL was similar. Accuracy of both assessments for correctly classifying patients who did or did not respond to EBV treatment, as measured using the HRCT-based TLVR response, was approximately three of four patients. The diagnostic performance of the Chartis System and HRCT fissure analysis were compared (Table?4). The sensitivity and specificity of the Chartis measurement were 86% and 61%, and those of HRCT fissure analysis 75% and 79%. The Chartis System and HRCT fissure analysis assessments were concordant in 47 patients (68.1%) and discordant in 22 (31.9%). In the discordant cases, the Chartis www.selleckchem.com/products/AC-220.html System correctly predicted TLVR response in 45% (10/22) and HRCT correctly predicted TLVR response in 55% (12/22) (Table?5). The clinical relevance of achieving TLVR ��350?mL with EBV treatment was assessed by calculating the pre- to post-procedure changes in clinical outcomes in patients dichotomized by TLVR response (